Gov’t Mule Shares Pro-Shot Video Of “The Man I Want To Be” From Capitol Theatre Concert Film [Watch]

first_imgNext month, Gov’t Mule will release a new concert film and live album, Bring On The Music, featuring pro-shot footage and recordings captured during the band’s April 2018 performances at The Capitol Theatre in Port Chester, NY. On Thursday, the band shared pro-shot video of “The Man I Want To Be” from their latest album, Revolution Come… Revolution Go. The newly shared video is the second release from the upcoming film, following “Life Before Insanity“.“The Man I Want To Be”[Video: Gov’t Mule]Bring On The Music – Live At The Capitol Theatre will feature more than two hours of video footage from the band’s 2018 performances in addition to interviews with the band, behind-the-scenes footage, photos, and more. Fans should click here to pre-order the live album and film ahead of its June arrival.Gov’t Mule also announced their anticipated return to Red Rocks Amphitheatre in Morrison, CO on August 18th, a new-found summer tradition following last year’s incredible super-sized show. Ryan Bingham will support. Pre-sale and VIP are underway now, and public on-sale begins May 31st. Gov’t Mule’s Summer of ’19 Tour kicks off June 21st and will feature support from newly announced openersKarl Denson’s Tiny Universe, The Record Company, Nikki Lane, and Joanne Shaw Taylor on select dates. For all ticketing details, please visit’t Mule is currently playing to packed houses overseas and will return to the States to headline Mountain Jam with a double-header performance on June 14th and 15th at Bethel Woods Center for the Arts. Warren Haynes will return as co-presenter of the festival for its 15th year.GOV’T MULE TOUR DATESMay 31 – Birmingham, United Kingdom @ Birmingham Town HallJune 1 – London, United Kingdom @ O2 Forum Kentish TownJune 2 – Manchester, United Kingdom @ Manchester Academy 2June 4 – Paris, France @ La CigaleJune 5 – Brussels, Belgium @ Ancienne BelgiqueJune 6 – Frankfurt Am Main, Germany @ BatschkappJune 8 – Raalte, Netherlands @ Ribs & Blues RaalteJune 9 – Alkmaar, Netherlands @ Podium VictorieJune 14-15 – Bethel, NY @ Mountain JamJune 21 – Cincinnati, OH @ PNC Pavilion at Riverbend^June 22 – Grand Rapids, MI @ Founders FestJune 23 – Indianapolis, IN @ The Lawn at White River**June 24 – Louisville, KY @ Iroquois Amphitheatre**June 27 – Boston, MA @ Rockland Trust Bank Pavilion**June 28 – Asbury Park, NJ @ The Stone Pony SummerStage^June 29 – Butler, OH @ Smoky Run Music FestivalJuly 3 – Asheville, NC @ Highland Brewing CompanyJuly 5 – Charlotte, NC @ The Fillmore (10th Anniversary Celebration)+July 6 – Greensboro, NC @ White Oak Amphitheatre+July 7 – Manteo, NC @ Roanoke Island Festival Park+July 10 – Charlottesville, VA @ Sprint Pavilion+July 11 – Baltimore, MD @ MECU+July 12 – Jay, VT @ Stateside AmphitheatreJuly 13 – Canandaigua, NY @ CMAC*July 16 – Toledo, OH @ Hensville ParkJuly 18 – Kansas City, MO @ Crossroads^^July 19 – Welch, MN @ Treasure Island Resort & Casino*July 20 – Walker, MN @ Moondance Jam*Aug 9 – Albuquerque, NM @ Isleta Amphitheater*Aug 10 – Oklahoma City, OK @ Chesapeake Energy Arena*Aug 18 – Morrison, CO @ Red Rocks Amphitheatre #Aug 27 – Saratoga, CA @ Mountain WineryAug 30 – Medford, OR @ Britt Music & Arts FestivalSep 4 – Salt Lake City, UT @ Red Butte Garden (On Sale May 6)Sep 5-8 – Las Vegas, NV @ Big Blues BenderSep 13 – Philadelphia, PA @ Outlaw Music FestivalSep 21-22 – Buffalo, NY @ Borderland Festival* w/ Lynyrd Skynyrd** w/ Karl Denson’s Tiny Universe+ w/ The Record Company^ w/ Nikki Lane^^ w/ Joanne Shaw Taylor# w/ Ryan BinghamWARREN HAYNES TOUR DATESJuly 24 – Westhampton Beach, NY @ Westhampton Beach PACJuly 25 – Westhampton Beach, NY @ Westhampton Beach PACJuly 26 – Newport, RI @ Newport Folk FestivalJuly 27 – Portsmouth, NH @ Prescott ParkJuly 28 – Scranton, PA @ Peach Music FestivalView Tour Dateslast_img read more

To clean up the mine, let fungus reproduce

first_imgHarvard-led researchers have discovered that an Ascomycete fungus that is common in polluted water produces environmentally important minerals during asexual reproduction.The key chemical in the process, superoxide, is a byproduct of fungal growth when the organism produces spores. Once released into the environment, superoxide reacts with the element manganese (Mn), producing a highly reactive mineral that aids in the cleanup of toxic metals, degrades carbon substrates, and controls the bioavailability of nutrients.The results, which will inform a wide range of future studies in microbiology, environmental chemistry, developmental biology, and geobiology, were published online this week in the Proceedings of the National Academy of Sciences (PNAS).Manganese is a versatile element, existing in multiple oxidation states and phases. Naturally occurring in the Earth’s crust, it plays essential roles in carbon sequestration, photosynthesis, and the transport and fate of nutrients and contaminants.“If you can get manganese to oxidize, then it forms these really active minerals, manganese oxides, which are environmental sponges that will clean up the water,” explains principal investigator Colleen Hansel (pictured), a faculty associate and former associate professor of environmental microbiology at the Harvard School of Engineering and Applied Sciences.It can be an especially important reactant in polluted water, such as the runoff from coal mines. When the ion Mn(II) is converted to higher oxidized states, it forms a reactive mineral that is extremely useful in getting other pollutants — such as arsenic, cadmium, and cobalt — under control and out of the water.“If you can get manganese to oxidize, then it forms these really active minerals, manganese oxides, which are environmental sponges that will clean up the water,” explains principal investigator Colleen Hansel, a faculty associate and former associate professor of environmental microbiology at the Harvard School of Engineering and Applied Sciences (SEAS). She is currently an associate scientist at Woods Hole Oceanographic Institution. “A lot of coal mine drainage remediation relies on getting bacteria and fungi to oxidize manganese to make these minerals.”“One problem with in situ remediation is that if you don’t know how and why processes are occurring, you can’t stimulate the organisms to do it. That’s been a big problem with the remediation of coal mine drainage sites. To stimulate microbial activity, the approach has been to provide complex carbon sources like corn cobs and straw and let the ‘bugs’ go to town, but it frequently doesn’t work.”It turns out that the common fungus Stilbella aciculosa only produces the necessary ingredient, superoxide, during cell differentiation (an aspect of growth and development) — specifically, during the formation of asexual reproductive structures. The finding implies that adding excessive nutrients to polluted water may not necessarily contribute to remediation, unless it is designed to induce fungal reproduction.For the fungus, superoxide appears to serve as a cellular signal that moderates cell differentiation. The chemical’s subsequent role in oxidizing environmental manganese so rapidly and efficiently may just be a useful coincidence, beneficial to humans but of little consequence to the fungus.All of the manganese-oxidizing bacteria and Ascomycete fungi known to date are heterotrophs; like humans, they eat carbon and breathe oxygen.“They’re not eating manganese the way some organisms eat other metals like iron,” says Hansel. “This has been an enigma, in the field of metal biogeochemistry. According to evolutionary theory, organisms usually perform a process for a reason. But for decades no one has understood why or how bacteria and some groups of fungi [the Ascomycota] were oxidizing manganese, because they weren’t doing it to gain energy.”Still, Hansel suggests that there may be more to the process than meets the eye.“It looks like an accidental side reaction, but we don’t really know, because manganese oxides are very reactive and could therefore provide some indirect benefits to the organism,” she says. “The manganese oxides could, for instance, degrade recalcitrant carbon and thus feed the fungi new carbon sources that they can metabolize better. Maybe they are ‘purposely’ doing it. We want to address these biochemical questions and the evolutionary implications, as well as determine the larger relevance of superoxide-based metal cycling. How important is this process in terms of the biogeochemistry of other metals like iron and mercury? How significant is its impact on the ecology of microbial ecosystems?”With co-authors Carolyn A. Zeiner (a graduate student at SEAS), Cara M. Santelli (a former postdoc, now at the Smithsonian National Museum of Natural History), and Samuel M. Webb (at the Stanford Synchrotron Radiation Lightsource), Hansel identified the biochemical mechanism that leads to the oxidation of manganese, including the class of enzymes (NADPH oxidases) that spur the process.The team’s discovery that superoxide is the key player in fungal oxidation of manganese is especially exciting because some bacteria actually do it the same way, even using the same enzymes. The idea that prokaryotes and eukaryotes developed this homology raises intriguing questions in the history of evolution.“We’re traveling down a whole new avenue in biogeochemistry,” says Hansel. “It’s exciting right now to be one of the people sitting in the front seat.”This project was funded by the National Science Foundation. The researchers also benefited from the resources of the SLAC National Accelerator Laboratory‘s Stanford Synchrotron Radiation Lightsource (SSRL) and the SSRL Structural Molecular Biology Program, supported by the Department of Energy (DOE) and the National Institutes of Health. A portion of the work was also performed using the DOE-supported Environmental Molecular Sciences Laboratory facility at Pacific Northwest National Laboratory.last_img read more

HIV treatment scale-up in rural South Africa shows dramatic results

first_imgThe large antiretroviral treatment (ART) scale-up in a rural community in KwaZulu-Natal, South Africa, has led to a rapid and dramatic increase in population adult life expectancy—a gain of 11.3 years over eight calendar years (2004-2011)—and the benefit of providing ART far outweighs the cost, according to new research from Harvard School of Public Health (HSPH).While previous studies have shown that ART significantly improves survival in clinical cohorts of HIV patients receiving ART, this is the first study to directly measure the full population-level impact of a public-sector ART program on adult life expectancy.“This is one of the most rapid life expectancy gains observed in the history of public health” said Till Bärnighausen, associate professor of global health in the HSPH Department of Global Health and Population and senior author of the study, which was published online in Science on February 21, 2013.“The public-sector scale-up of ART has largely reversed the decline in adult life expectancy due to HIV that occurred in the 1990s and early 2000s in the region,” said Jacob Bor, the lead author of the study and an HSPH doctoral candidate in the Department of Global Health and Population.The researchers measured dates of death using data from a large community-based population surveillance system that included information on all births and deaths among more than 100,000 people living in rural KwaZulu-Natal, in South Africa, between 2000 and 2011. Read Full Storylast_img read more

FACES 2011 review

first_imgAnother year quickly ends. And we thank you, our Georgia FACES subscribers, for allowing us to bring you the news to use about Georgia family, agricultural, consumer and environmental sciences.The news service for the University of Georgia College of Agricultural and Environmental Sciences, Georgia FACES generated more than 300 news items this year, releasing them across the state, region and nation. On behalf of the writers and their helpful sources, I wish you and your family a Merry Christmas and prosperous new year. It’s been a pleasure.To honor the passing year, we look back on 2011’s top 11 FACES news stories listed by publication date. Why 11? Because it is one more than the typical top 10, and FACES believes in delivering more than the typical.Georgia eyes coldest winter everUGA trains National Guard for ag mission UGA weather monitoring network struggles to survive Sustainable agriculture: Two sides of the same coin Drought tightens grip on Georgia farmersUGA study investigates impact of farm labor shortagesKudzu bug spreads across Southern statesGeorgia’s summer was, in a word, “Hot!”UGA training helps former homeless men prepare for jobsPig induced pluripotent stem cells may be safer than previously thought What is the right way to say ‘pecan’?last_img read more

EIA estimates U.S. coal production down almost 20% year to date compared to 2019

first_img FacebookTwitterLinkedInEmailPrint分享S&P Global Market Intelligence ($):Total U.S. coal production for the week ended April 18 decreased 41.5% year over year to 8.6 million tons from 14.7 million tons, according to data from the U.S. Energy Information Administration.For the 52 weeks ended April 18, production was 658.7 million tons, representing a year-over-year decline of 11.9%, while year-to-date coal output slipped 19.6% year over year to 173.6 million tons.The western region’s coal production for the week reached 4.6 million tons, posting a 40.4% decrease from the prior year’s 7.8 million tons. Data for the western region covers Powder River Basin mines.Coal production from Appalachian mines totaled 2.4 million tons, declining 43.6% from the year-ago week’s 4.3 million tons.The interior region’s production decreased 41.3% to 1.6 million tons, compared to 2.7 million tons a year ago. Interior region data covers mines in the Illinois Basin.[Jacob Holzman]More ($): U.S. weekly coal production down 41.5% YOY EIA estimates U.S. coal production down almost 20% year to date compared to 2019last_img read more

Behind the Scenes at an Ebola Outbreak

first_img#452890086 / Another reason the outbreak I witnessed was contained so well was that the message from the Ugandan Ministry of Health, from Lacor Hospital, and from all the international responders, was clear and consistent: Avoid bodily fluids. Do not continue with traditional funeral ceremonies that involve cleansing of the dead body. If you are sick, go to the doctor so you don’t get your loved ones sick.The message was on radio stations, announced from PA systems mounted on cars driving up and down roads, posted everywhere. I have a T-shirt with a person holding another person who is vomiting, all in a circle with a big X through it—don’t touch bodily fluids. The suits gave a feeling of otherworldliness. I always felt calm and focused in the MCL. And I never worried. I was hard-pressed to think of a way I could possibly become contaminated with Ebola virus. I didn’t do any work with animals, so I never had to use a needle, and Ebola is not transmitted by air, only by fluids—and those fluids would have to be in contact with my own bodily fluids to get me sick. Everyone who worked in the MCL knew that we were safe. Part of the safety training even involved a discussion of what to do if another researcher in the lab fell to the ground and needed help. We all knew we would open his or her suit as well as our own in a second to do CPR. There was no Ebola floating around in the air. It doesn’t do that. It just wasn’t a concern.So when Stuart asked me if I would be willing to go to Uganda as part of the team that was to replace our experts who had already been in country for several months, I talked it over with my husband and told Stuart yes the next day. I wasn’t particularly excited about the opportunity (still ridiculously worried about the size of the bugs!), but I wasn’t at all worried about getting Ebola virus.I’m pretty sure I flew out on December 19, 2000, or else I arrived in Uganda or traveled up north on the 19th. That’s my father’s birthday and I remember thinking it wasn’t much of a present to have his daughter traveling to northern Uganda. I was stationed at St. Mary’s Lacor Hospital just outside of Gulu, Uganda. And as if Ebola virus wasn’t enough for my poor husband, parents and brothers to worry about, there was ongoing rebel activity in the area. #1320445 / Sign up for our COVID-19 newsletter to stay up-to-date on the latest coronavirus news throughout New York [dropcap]A[/dropcap]nyone who has worked in a maximum containment lab with Ebola virus has had that moment when a headache starts and you wonder: “Is this it? Did I do something wrong in lab? Did I infect myself?” Thankfully, that moment is short-lived; it is just a normal human reflex to worry about such a thing, but the truth is that rationally we all know that we didn’t infect ourselves.Short of a prick with a needle full of virus, it is pretty much impossible for us to imagine how we would get infected with Ebola virus in a maximum containment lab. Actually, it is pretty difficult to imagine getting infected out in the real world, too. That is why the people who work with the virus are always willing to go to outbreaks. It is why, when my boss asked me to travel to Uganda in 2000 to be part of the International Ebola Response Team there, I didn’t worry about anything other than what size bugs might confront me. Seriously. It sounds crazy or stupid, but it is true. #1320421 / With today’s air travel it is completely possible that a person with Ebola virus will enter the United States. But this virus is just not that easily transmitted. A person infected with it, but not yet showing symptoms, cannot pass the infection on to someone else. Once they have symptoms they can, but still, the virus is not airborne. And we don’t have a tradition in the U.S. of washing dead bodies at home, or of welcoming friends and family of the deceased to take part in such a ceremony. So overall, I see the chance of a person in the U.S. passing an Ebola infection to a large number of people as astronomically low. And while a single person being infected and/or passing that on to another single person is scary and desperately sad, it is really no scarier, no sadder than the limited transmission of any other high-mortality infectious disease in our country.[dropcap]I[/dropcap] still study Ebola virus at the lab bench, but now just a small, noninfectious part of it. I am still fascinated by it. After having been to an outbreak, I try to be more cognizant that it is not just a fascinating virus but also a life-changer for whole communities. Lacor Hospital and the people of northern Uganda suffered enormously as a result of Ebola virus. Just as I am certain that people living in western Africa, and people who live in the U.S. but love people living there, must be suffering, both physically and emotionally now.In the case of the Ugandan outbreak, I would like to think that some good came of it.There was a lot of international press about the outbreak and about Lacor Hospital. It was already the recipient of numerous international healthcare and humanitarian awards, but more still came in afterwards. They were finalists for the Hilton Humanitarian Prize in 2007 because they deserved it, and since I had been there and witnessed its excellence I wrote the nomination letter for Lacor. In 2001, USAID began distributing the funding promised by Secretary of State Madeleine Albright to Lacor Hospital. These funds built the beautiful new children’s ward and laboratory facilities at the hospital. In 2013, the nurses of Lacor won the prestigious Sigma Theta Tau Nursing Society Award.And I hope that the founding of Social Promise, the nonprofit that I direct, will continue to provide Lacor Hospital with funds to operate for years to come.Sharon McGee Crary is the Percy L. Julian Associate Professor of Chemistry and Biochemistry at DePauw University & Co-founder and President of the Board of Directors at Social Promise, a nonprofit that partners with Ugandan nonprofits to help provide critical health and educational resources to their local communities. She traveled to northern Uganda in 2000 as a member of the Viral Special Pathogens Branch at the Centers for Disease Control and Prevention to help diagnose cases of Ebola hemorrhagic virus—the often fatal illness currently gripping West Africa—during a deadly outbreak that claimed more than 400 lives. I joined the Viral Special Pathogens Branch at the Centers for Disease Control and Prevention in Atlanta, because I loved studying RNA—biological molecules vital in human genetics. But I was interested in doing research that had a bit more of a human aspect to it than the enzyme kinetics stuff I had done in graduate school at Duke. So an RNA virus like Ebola virus seemed like the perfect choice. Its categorization as a Level 4 pathogen, combined with the fact that it was only identified in 1976, meant that not much was known about its molecular mechanisms yet. So there was plenty of work to be done.During my interview for the position, I remember being at dinner with my future boss, Dr. Stuart Nichol, along with Dr. Jonathan Towner, who was a postdoc in the lab at the time (he has since become a full-time employee of the CDC and is often in the frontline of outbreak responders). Stuart explained to me that it was unlikely I would be sent to an outbreak; they were not that common and everybody wants to go into the field. That was fine by me. It never occurred to me that someone would want to be sent to Africa to respond to an outbreak; I just wanted to stay at the lab bench.And I loved working in the lab at the CDC, especially the maximum containment lab (MCL). Everything about working there was perfectly organized and calm and intelligent. My bosses in the Special Pathogens Branch—Drs. Stuart Nichol, Tom Kziasek and Pierre Rollin—had made sure of it. The way we entered and exited the lab was perfectly scripted to keep us and the outside world safe. On the way in, there were well-thought through plans for the order and placement of changing into scrubs, sealing socks and gloves with tape, inserting ear plugs, putting on Biosafety Level 4 suits, hooking up to oxygen, going through the airlock, and cleaning the contaminated airspace behind us by running the decontamination shower and pulling on protective boots.Once inside, everything was equally regimented. The first one in each day had to run through the checklist to make sure all the lab equipment was still working. But whenever you entered, it was always: hook up to oxygen, walk forward and sign in, turn right and check that the refrigeration units were working properly, and so on. In the MCL we were immediately surrounded by calm. We wore ear plugs to protect our hearing from the sound of the oxygen rushing into our suits, but we could still hear it—like a white noise machine. That meant it wasn’t easy to talk to other researchers in the lab, which made the time in the MCL private and centered.center_img [dropcap]I[/dropcap]n Uganda, our “maximum containment lab” was a bit more primitive than at CDC headquarters in Atlanta. There were no submarine doors or decontamination showers, but there were portable personal protection suits and spray bottles full of Jik—the Ugandan brand of bleach—and a tray full of Jik to step through upon leaving the lab. And this is enough. As long as you don’t touch Ebola virus or get large droplets of it in your eyes or mouth, you won’t get infected.Even the doctors treating the Ebola patients just use this basic barrier protection—the same outfit worn by the person in the screening tent outside of isolation. The boots protect feet against any potentially sharp objects (piercing the skin with an object contaminated with Ebola virus would be dangerous), the apron, gloves, mask and goggles protect the wearer from contact with Ebola virus and are all sprayed down with Jik upon leaving the ward, and the scrubs protect the street clothes underneath.All the layers and protective equipment come off when a person leaves the lab or isolation ward. They are all discarded and incinerated, or completely decontaminated with Jik and washed. Ebola is easy to kill. It doesn’t like bleach or hospital-grade Lysol, so basic measures protect against transmission in a hospital setting.By the time I got to Gulu, the outbreak was winding down. There were still people in isolation and I ran around 20 diagnostic PCRs each day for most of the 40 days I was there, but no one died at Lacor during that time.The first team saw the worst of it and it was bad. It was the largest outbreak of Ebola virus up to that time and many people died. Lacor Hospital lost too many personnel, including Dr. Matthew Lukwiya, the doctor who was supposed to take over as the first Ugandan director of the hospital (New York Times Magazine published a beautiful article about him). No one ever really knows how the hospital infections of doctors or nurses occur, but it seems to be related to mistakes. It is awful to say this, because no one wants to know that a death was preventable, but on the other hand, it means that these infections and deaths don’t have to happen, and that is good.In general, I would say that hospital personnel in the U.S. have extensive experience wearing gloves, masks and goggles. They are used to wearing them and know how to avoid self-contamination. I know this from working in lab. When I wear gloves I would never touch my face. I take off my gloves in a certain way to avoid touching the outside of them. I never lift goggles from my face to rub my eyes. These are habits that form over years of wearing protective equipment. Perhaps at Lacor Hospital the staff was not yet as adept at these habits. Plus, they were working in brutally hot conditions, under great emotion stress. On several occasions the nurses at Lacor thought about leaving the isolation ward but some always stayed, inspired by Dr. Matthew’s leadership. Maybe this is the same thing that happened to the American doctor and nurse who were recently infected. Maybe they were just dealing with the current epidemic under less-than-ideal conditions for too long. Maybe they didn’t have the supplies or protective equipment that they needed. Maybe the nonstop emotional stress allowed them to make one wrong decision. #481844265 / Gulu was the epicenter of the horrendous war waged by the Lord’s Resistance Army throughout the last decades of the 20th century and into the 21st. But it turns out rebels worry about Ebola virus, too—so they weren’t around while I was there. It was a strange thing to think that there was an upside to an outbreak of Ebola hemorrhagic fever.There are a few different types of Ebola virus. The one I worked with most often back home in lab was called Zaire. It was originally identified in what is now the Democratic Republic of the Congo in 1976. It is this same virus that is infecting people in western Africa right now. And unfortunately, it is the type that causes the most death, with a fatality rate of about 90 percent. I felt lucky at the outbreak I was a part of because it was caused by Ebola Sudan, which has only a 50-percent fatality rate. Compared to Zaire, that seemed like good odds, which was good the one time I did feel a little sick while I was in Uganda. I figured that 50/50 were decent odds. But, just like back home, I knew rationally that I didn’t have Ebola. I was in charge of molecular diagnostics for the isolation ward at Lacor Hospital. This meant that I ran PCR (polymerase chain reaction) on blood samples that were already decontaminated by other lab workers.Here’s how isolation worked during our outbreak. (The protocol is always the same, at every outbreak, because a lot of the same doctors show up to each outbreak due to there being so few experts in this field. Additionally, because this is the best way to prevent the spread of Ebola. So it will be the same at an isolation unit in western Africa right now. And it would be the same in the U.S. if needed.)A person presents to the isolation ward and answers a series of questions posed to them by a person wearing scrubs, boots, gloves, goggles, a mask, one of those surgical hats that doctors wear, and an apron. The questions range from how the patient feels to whether anyone they have been in contact with was sick with Ebola. If a person has certain characteristics, such as a high fever with other concurrent symptoms, unexplained bleeding, or fever and contact with a person who was known to have Ebola hemorrhagic fever, they are admitted to the isolation ward. Once inside, a doctor sees the patient and can order blood to be drawn for testing if s/he thinks the patient should remain in the isolation ward. The blood is sent to the diagnostic lab. (In our case this lab was onsite at the same hospital, but that is not likely to be the case in western Africa right now given the geographical range of the outbreak. It also depends on the level of facility at the site of the isolation ward, and we were lucky to be at such a fine hospital with excellent lab facilities for sub-Saharan Africa.)There are two tests used to confirm Ebola infection: an immunological test and a molecular test. The molecular test was first introduced during the outbreak I was part of, by Jonathan Towner—the postdoc who was in the first team to respond to that outbreak. The blood goes first to the people who do the immune assays—tests that can detect specific proteins—and they were the ones who would decontaminate a sample to hand out to me for the molecular diagnostics. But I know that must be hard. And that is a concern.[dropcap]I[/dropcap] think about what I do when one of my sons is sick. I cuddle them and kiss them (even though they are getting too old for that, they can’t protest as much when they are sick!). I wipe their feverish brows with damp cloths, and yes, I hold containers for them to vomit into and then I clean up. What if one of them had Ebola? It is definitely possible, although not guaranteed, that I would get infected from him. Which is why it is so important that I know if there is any chance he is suffering from Ebola. Given that the bats that are thought to harbor Ebola virus don’t live near me, and we don’t know anyone who has traveled to western Africa lately, I think the chances are essentially zero. But, if we did know someone who had just returned from one of the countries currently suffering with the outbreak, and that person had come to our home, then got sick, and then one of my sons got sick, I would call my doctor and tell her all those details. And I think I would end up bringing my son to a hospital where he might be put in isolation while tests were run on him. That would be extraordinarily difficult. I don’t know if here in the United States I would be allowed to suit up and go sit near him. I sort of doubt it. I’m sure I wouldn’t be allowed to do so if it were my husband who was sick, but maybe for a child? But it would be the only certain way to protect my other son and my husband and our friends and family from infection.I do worry a little about these things—about the likelihood that U.S. citizens would subject themselves to contact tracing or hospital isolation. These are the main weapons we have against an epidemic of Ebola hemorrhagic fever anywhere, and it does concern me a bit that here, where we have the best hospitals and doctors trained to handled just such an infection, we might hesitate to use them for selfish reasons. But I also have hope that we will. Because I know the types of people who are involved in the tracing and in running the isolation wards. They are good, kind people who truly care. They are intelligent and patient. In the end, I do have confidence that we will rely upon our experts if that necessity ever arises.But I have gone off on a tangent, talking about what would happen in the case of an actual epidemic, because I am actually not worried that there will ever be a full-out outbreak of Ebola virus in the U.S. Maybe the fact that I talked about what it would look like anyway will help convince you that the people who work with Ebola virus are prepared for any scenario.They are thinkers. They are planners. They have a worse-case scenario plan in mind that takes into account the fact that we are human beings who deserve to be treated with respect and love and thoughtfulness. But they also realize that really, this is a plan that will likely never be implemented. [dropcap]T[/dropcap]his type of outbreak will never happen in the United States, in my opinion. And I am sure that anyone else who has experience with Ebola virus will tell you the same thing. Doctors and nurses in the U.S. already deal with infectious diseases that are much more easily spread than Ebola, without getting sick and without transmitting the virus to other patients. So once a patient with Ebola hemorrhagic fever is in one of our hospitals, containment should be simple: gloves, Lysol, masks. We even have special hospital units with glass windows protecting visitors from patients. We have procedures in place for autoclaving waste and decontaminating surfaces. Our hospitals are ready for this. Ebola just doesn’t get transmitted that easily from person to person.So is there anything I am worried about? And why do I think the outbreak in western Africa is so big and has been going on for so long? I’m worried that citizens in the U.S. would never be as forthcoming about turning themselves in for isolation as the people in northern Uganda were. And I think this is the crux of the problem in western Africa today.Remember I said the path to isolation starts when a person presents to the isolation ward? Well, that step is essential. The reason I’m not worried about the two people being treated for Ebola virus in the United States right now is that they are in a hospital being cared for by experts. Not only does it increase their chance of fighting the disease—because they will get the best support possible—but it decreases the chances of anyone they love getting the disease.These two did the right thing. When they thought they had Ebola they followed the normal procedures and went into isolation. At Lacor Hospital, I saw the same thing. People there trusted their hospital. Lacor Hospital was one of the only graces in that rebel war-torn area for decades. The hospital wasn’t just a place to go for superb medical care, it offered a safe place for children to sleep at night to avoid abduction by the rebels. The people of northern Uganda knew that Lacor cared for them. They trusted that the doctors, nurses, and directors of Lacor would do what was best for them, just like they always had in the past. I suspect the same is not true throughout the afflicted countries in western Africa. We have all heard reports of people there fleeing from health care workers. And that is certainly one reason it is so difficult to get that outbreak under control.I hope the same thing would never happen in the United States, but I suppose it is possible.If a person was found to be infected with Ebola here in the U.S., teams of epidemiologists would be assigned to track down every single person who had any recent contact with that patient. Each of these ‘contacts’ would be questioned, in person, every day, about any potential symptoms of illness. This would go on for 21 days—the longest time that Ebola virus has ever been found to live inside a person. And at the end of that time, if no one else got sick, we would all be safe. If one of the contacts did get sick, he would be hospitalized and all of his contacts would be traced for 21 days.In the U.S., we have the infrastructure and capacity to do this. And our hospitals are used to containing diseases, especially those that are not airborne and are easily decontaminated. But I do worry a bit that people would not take well to being tracked by government employees for 21 days. Or that they would refuse to bring themselves to the isolation ward. I was always impressed that the people in Uganda did that so willingly, knowing they might never see their loved ones again. But they took to heart the fact that it was the best way to protect these loved ones from the same fate.last_img read more

3 tips for improving your non-verbal communication skills

first_img 43SHARESShareShareSharePrintMailGooglePinterestDiggRedditStumbleuponDeliciousBufferTumblr,John Pettit John Pettit is the Managing Editor for John manages the content on the site, including current news, editorial, press releases, jobs and events. He keeps the credit union … Web: Details From the minute you shake someone’s hand for the first time, communication is important. The words you use are vital, but if you want to be sure to make the right first impression (or hundredth) you should always be mindful of the non-verbal message that you’re conveying. Here are three ways you can improve your non-verbal communication skills.Be aware of your body: As a leader, you probably talk a lot. Make it your mission to be mindful of the message your body is sending. You may have something else on your mind, but when you’re speaking with an employee or colleague, you want them to know that you’re focused on the present. When you’re speaking, your listener is probably looking at your face, so be especially sure to avoid any negative reactions (frowns, eye-rolling, etc…), but also be mindful of the rest of your body. Slouching or turning away will let someone know you don’t care about what they have to say. Don’t be too exaggerated, but make your body language match up with the words that you’re saying.Keep your eye on the target: You’ve never heard that eye contact is important. Obviously, I’m kidding, but in the device-filled world we live in, verbal communication has taken a backseat to text messages. I haven’t heard of anyone proposing to their significant other via iMessage, but I’m sure it’ll happen eventually (if it hasn’t already). Face-to-face communication is a lost art, but for those of us that still enjoy it, it’s important to keep an eye on your target so they know you’re fully committed to the conversation.Keep your tone positive: Sometimes you’re told things that you couldn’t possibly care less about. When someone is looking for approval or another positive reaction, try to avoid a tone that is dull and uninterested. When others are excited about news they’re sharing, you should come across as excited, or at the very least happy to hear the news. Think about the ways you’ve responded in the past and do your best to use a proper tone when talking to employees and coworkers.last_img read more

Jacob’s Pillow Theater, Site of Dance Festival, Destroyed in Fire

first_img– Advertisement – A theater at Jacob’s Pillow, a destination for dance performance in Massachusetts, was destroyed on Tuesday in an early morning fire.The fire was reported around 7 a.m. at the Doris Duke Theater in Becket, Mass., according to a statement from Jacob’s Pillow. Videos from the scene showed a collapsed building engulfed in smoke with firefighters blasting water onto heaps of charred wood. The damage to the theater was extensive, the statement said, but was contained to the one building.- Advertisement – The cause of the fire is not yet known. The theater that was destroyed is the newer one of the two on campus. It opened in 1990 as a smaller, more informal space compared to the main Ted Shawn theater, which opened in the 1940s. The Doris Duke Theater had about 230 seats and a back wall that could open to the surrounding wooded area. It was created as a space intended for up-and-coming companies to perform.center_img The performance space is one of two indoor theaters at Jacob’s Pillow Dance Festival, an annual summer event that attracts some of the world’s leading companies. The festival was canceled this year because of the pandemic. – Advertisement –last_img read more

Anies hints at ‘pulling brake’ as Jakarta cases continue to surge

first_imgJakarta, the country’s first epicenter of the COVID-19 outbreak, is mulling whether to reimpose restrictions it previously eased as the daily number of new cases hits new records.Jakarta reported 281 new cases on Monday, making a total of 14,797 cases and 697 deaths so far. This figure was among the city’s record daily highs since the outbreak hit, the fifth-largest after 404 on Sunday, 378 on Saturday, 357 on Wednesday and 284 on Thursday.The latest daily records came a day after Jakarta Governor Anies Baswedan said the city might consider tightening its large-scale social restrictions (PSBB). Jakarta has gradually relaxed restrictions since June 4 in the hope of easing economic suffering, with businesses and offices reopening under new health protocols. It has also reopened public green spaces and outdoor tourist destinations. “If this continues, we may have to return to [social restrictions]. […] Do not let this situation reach the point where we have to pull the emergency brake,” Anies said on Sunday.The emergency brake policy would see a restoration of stricter PSBB, which had allowed only eight essential sectors to operate during the pandemic.“If that happens, we all have to go back home, and economic, religious and social activities will be halted,” Anies said.He acknowledged the persistent outbreak in Jakarta, saying that while most of the new cases were the result of the administration’s massive “active case finding” efforts, the city’s positivity rate — the number of people who test positive divided by the total number tested — has caused concern. Jakarta’s weekly positivity rate had consistently been below 5 percent since the beginning of June, in line with one of the World Health Organization’s requirements for regions seeking to enter the so-called “new normal”.However, the positivity rate on Saturday was recorded at 7.1 percent and 9.6 percent on Sunday, with a weekly average of 5.7 percent last week.“That is why I want to tell all Jakartans: do not take this lightly. Do not feel like we are free of the COVID-19 outbreak,” Anies said.Jakarta authorities said 45.2 percent of the total cases were patients taking tests in hospitals, 38.4 percent were people taking tests in residential areas, including people living in densely populated areas and areas with high incidence rates, and 6.8 percent in wet markets, including among vendors.With the outbreak showing no signs of abating, Anies warned the public to maintain their daily routines with extra caution in vulnerable hot spots: public transit and wet markets.The increasing number of daily cases in Jakarta, one out of eight provinces with similar trends, has gained the attention of President Joko “Jokowi” Widodo, who, on Monday, reportedly said that reimposing restrictions had always been on the table.“The President has always reminded [authorities] to pull the brake proportionally,” national COVID-19 task force chief Doni Monardo said on Monday. “If cases surge, then pull the brake. But not all activities should be completely suspended. It needs to be limited in terms of timing of activities and number of people involved.”Hermawan Saputra from the Indonesian Public Health Expert Association (IAKMI) criticized Jakarta’s easing of restrictions on activities not directly related to fulfilling primary needs.”[A policy] has to be flexible to a certain degree. In terms of the pandemic, it can be applied to economic activities aimed at ensuring the fulfillment of basic necessities, efforts to encourage the informal sector and medium businesses, but not all activities,” he said.Jakarta’s recent move to reopen tourism spots and weekly events like the Car-Free Day was therefore unnecessary, Hermawan said.Hariadi Wibisono, the Indonesian Epidemiologists Association (PAEI) chairman, said the Jakarta administration should not hesitate to reimpose restrictions if necessary, saying such a U-turn “is part of decision making and not a display of inconsistency because every decision needs evaluation”.Epidemiologist Pandu Riono from the University of Indonesia’s School of Public Health said, however, returning to strict restrictions without stern enforcement of health protocols was not a solution to the new transmission problem.”If we go back [under the stricter PSBB] but people continue to fail to abide [by the restrictions], it would not change anything much,” he said.In addition, he called for cities in Greater Jakarta to implement strict health protocols as many commuters might carry the virus without symptoms.The experts said the government should also involve local figures in promoting the importance of implementing health protocols and in reaching out to the wider public to prevent misconceptions that mask-wearing and social distancing were no longer needed.Topics :last_img read more

Pay at Canadian pension managers ‘completely, utterly unacceptable’

first_imgIn a survey conducted for his book, Ambachtsheer found that, across 10 Canadian pension funds, more than 2,160 full-time employees responsible for $631bn (€476.1bn) in assets earned $448m at the end of 2010 – an average of $207,000 per person.In comparison, 10 US funds managing $902bn employed 1,469 full-time staff for $187m, or $127,800 on average, below the average $157,100 paid to 579 staff across eight funds in Northern Europe and the UK.Holmes said she was hopeful the UK had a sufficiently large number of pension funds against which to benchmark pay to avoid a situation similar to Canada.But she struck a cautious note due to the increasing number of international pension funds setting up London offices, saying there was no easy answers on the matter of pay.Canadian pension plans that have opened London offices have “a bunch of empty desks” and would look to institutions including the £49bn (€66.9bn) USS and £23bn Railpen Investments to hire private market staff, she said.“When we talk about [remuneration] at USS, we talk about it in a very different way than [they do at investment banks] because we believe people are privileged to work at USS, not least because they don’t have to go out and sell,” she said. For his part, Ambachtsheer argued that pension fund investment was a “high-compensation market” and that the model in Canada was often for base salaries of CAD500,000, with a performance-related element enabling a three or fourfold increase over the base.A similar approach is employed by the CAD282bn Canada Pension Plan Investment Board, where the six highest-paid executives, including chief executive and president Mark Wiseman, had a combined base salary of CAD2.5m in 2014 but total remuneration packages worth CAD19.5m.This compares with the highest-paid individual at USS IM earning £991,000 in 2014, according to the manager’s most recent accounts. Ambachtsheer noted the funds in Canada needed to balance staff ability to earn multiples of their income in the private market versus the comparatively lower pay of those contributing to many of the funds. Read a review of Ambachtsheer’s book ‘The Future of Pension Management’Some quotes have been modified since publication The chair of the Universities Superannuation Scheme’s (USS) in-house manager has criticised the “completely and utterly unacceptable” levels of pay at Canadian pension funds, warning of the impact on the UK pensions market.Virginia Holmes – who, in addition to chairing USS Investment Management (USS IM), is a board member of the CAD90bn (€61bn) Alberta Investment Management Corporation (AIMCo) – said USS was questioning how to retain its staff with Canadian investors setting up London offices.“When I go across to Canada and [meet remuneration consultants], I am truly and genuinely shocked, and I take a lot of being shocked,” the former UK chief executive of AXA Investment Managers said.Speaking at the book launch of Keith Ambachtsheer’s ‘The Future of Pension Management’, she added: “You seem to have this classic scenario [in Canada] where seven funds have benchmarked themselves up and up and up to levels that are completely and utterly unacceptable in the public purpose world.”last_img read more